Arrhythmias

Why Antiarrhythmic Drugs Are High-Risk Drugs
Connect cardiac electrophysiology and rhythm assessment with antiarrhythmic drug mechanisms, clinical selection, monitoring, and prevention of proarrhythmic harm.

Course Content

01. Why Antiarrhythmic Drugs Are High-Risk Drugs
02. Normal Cardiac Conduction
03. ECG Intervals: What Drugs Change
04. Ventricular Action Potential and Ion Currents
05. Nodal Action Potential and Autonomic Control
06. Mechanisms of Arrhythmogenesis
07. First Clinical Decision: Stable or Unstable?
08. Vaughan Williams Classification
09. Class I Shared Biophysics
10. Class I Subgroups: Kinetics and ECG Effects
11. Class IA: Quinidine, Procainamide, Disopyramide
12. Class IB: Lidocaine and Mexiletine
13. Class IC: Flecainide, Propafenone, and CAST
14. Class II: Beta Blockers
15. Class IV: Non-DHP Calcium Channel Blockers
16. Adenosine: The AV Node Reset Drug
17. Class III: Potassium Channel Blockers
18. Reverse Use-Dependence
19. Amiodarone: Broad-Spectrum Multi-Channel Drug
20. Amiodarone Toxicity and Monitoring
21. Other Class III and Amiodarone-Related Drugs
22. Magnesium, Electrolytes, and Torsades
23. Digoxin: AV Nodal Drug and Toxicity Trap
24. Bradyarrhythmia Pharmacology
25. Atrial Fibrillation and Flutter: Framework
26. WPW and Pre-Excited Atrial Fibrillation
27. PSVT: AV Node-Dependent Tachycardia
28. Wide-Complex Tachycardia and Ventricular Tachycardia
29. Comparative Safety Matrix
30. Final Board-Ready Drug Selection Map
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